Publications
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2012. Disassembly of simian virus 40 during passage through the endoplasmic reticulum and in the cytoplasm.. J Virol. 86(3):1555-62.
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2006. Simian virus 40 late proteins possess lytic properties that render them capable of permeabilizing cellular membranes.. J Virol. 80(13):6575-87.
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1999. Extracellular simian virus 40 transmits a signal that promotes virus enclosure within caveolae.. Exp Cell Res. 246(1):83-90.
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1999. Simian virus 40 infection via MHC class I molecules and caveolae.. Immunol Rev. 168:13-22.
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1998. MHC class I molecules are enriched in caveolae but do not enter with simian virus 40.. J Gen Virol. 79 ( Pt 6):1469-77.
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1996. Bound simian virus 40 translocates to caveolin-enriched membrane domains, and its entry is inhibited by drugs that selectively disrupt caveolae.. Mol Biol Cell. 7(11):1825-34.
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1996. Extracellular simian virus 40 induces an ERK/MAP kinase-independent signalling pathway that activates primary response genes and promotes virus entry.. J Gen Virol. 77 ( Pt 9):2173-82.
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1996. Multiple stages of virus-receptor interactions as shown by simian virus 40.. Adv Exp Med Biol. 408:159-67.
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1995. Relationship between expression of epidermal growth factor and simian virus 40 T antigen in a line of transgenic mice.. Transgenic Res. 4(5):306-14.
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1992. Class I major histocompatibility proteins are an essential component of the simian virus 40 receptor.. J Virol. 66(4):2037-45.

Department of Microbiology