Drupal-Biblio17<style face="normal" font="default" size="100%">Association of caveolin with Chlamydia trachomatis inclusions at early and late stages of infection.</style>Drupal-Biblio17<style face="normal" font="default" size="100%">Caveolae in the uptake and targeting of infectious agents and secreted toxins.</style>Drupal-Biblio17<style face="normal" font="default" size="100%">Extracellular simian virus 40 transmits a signal that promotes virus enclosure within caveolae.</style>Drupal-Biblio17<style face="normal" font="default" size="100%">Simian virus 40 infection via MHC class I molecules and caveolae.</style>Drupal-Biblio17<style face="normal" font="default" size="100%">MHC class I molecules are enriched in caveolae but do not enter with simian virus 40.</style>Drupal-Biblio17<style face="normal" font="default" size="100%">Bound simian virus 40 translocates to caveolin-enriched membrane domains, and its entry is inhibited by drugs that selectively disrupt caveolae.</style>Drupal-Biblio17<style face="normal" font="default" size="100%">Extracellular simian virus 40 induces an ERK/MAP kinase-independent signalling pathway that activates primary response genes and promotes virus entry.</style>Drupal-Biblio17<style face="normal" font="default" size="100%">Multiple stages of virus-receptor interactions as shown by simian virus 40.</style>Drupal-Biblio17<style face="normal" font="default" size="100%">Relationship between expression of epidermal growth factor and simian virus 40 T antigen in a line of transgenic mice.</style>Drupal-Biblio17<style face="normal" font="default" size="100%">Virus receptors: implications for pathogenesis and the design of antiviral agents.</style>Drupal-Biblio17<style face="normal" font="default" size="100%">Class I major histocompatibility proteins are an essential component of the simian virus 40 receptor.</style>